The Pathogen Hypothesis
J Pathol. 2008 Sep 18. Abstract References [1] Balin BJ, Gerard HC, Arking EJ, Appelt DM, Branigan PJ,
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Early Detection of Alzheimer Disease—A Virtual Town Hall Meeting
disease. Q: Does anyone believe PET imaging, perhaps with F-18 amyloid tracers, will be clinically
Targeting Tumor Necrosis Factor—A Therapeutic Strategy for AD
affected the clinical status of patients with moderate to severe heart failure (18-20). Etanercept (Enbrel)
APP Family: Clues to Function in Health and AD
2007 Sep 15;120(Pt 18):3157-61. No abstract available. Abstract 0 NS: Text requested September 12. NS:
Susceptibility Testing and Risk Assessment in Alzheimer Disease
1999. 3: p. 65-70. Abstract 18. Sadovnick, A.D. and S. Lovestone, Ethical and legal issues: Genetic
Presenilin Loss of Function—Plan B for AD?
These are comfortable times for the amyloid hypothesis, it would seem. Every week brings more good news about some anti-amyloid intervention having “cured” mice from their “Alzheimer’s.” On the human front, we are eagerly awaiting such therapeutics to sho
The Functional Roles of APP Cytoplasmic Domain—Conflicts and Consensus
Atlanta (Tuesday, October 18, 8:30 a.m.). Some of the items up for discussion will include the following:
Whole Genome Study for Parkinson Disease
Lancet Neurol. 2006 Nov ;5(11):911-6. Abstract Katrina Gwinn-Hardy led this live discussion on 18
Messing with the Membrane—An Alternative Interpretation of the Amyloid-β Hypothesis
We invite you to participate in this "offline" Forum discussion led by Vincent Marchesi of Yale University. Coming from a different research field, Marchesi has in recent years followed the AD literature as closely as have few other outside obse
Cell Cycle Hypothesis Pedaling into Mainstream Acceptance? Results in Fly, Mouse Models Warrant a Second Look
Vikram Khurana, Karl Herrup, Bruce Lamb, Inez Vincent, Rachael Neve, Donna McPhie, Dan Geschwind, Cathy Andorfer, and Xiongwei Zhu participated in a discussion of how far the cell cycle hypothesis has come in the past few years, and where to go next. Vik
Gain or Loss of Function—Time to Shake up Assumptions on γ-Secretase in Alzheimer Disease?
We invite you to participate in this “offline” Forum discussion with past ARF advisors Peter Davies of Albert Einstein College of Medicine in the Bronx, New York, and Bart De Strooper at K.U. Leuven, Belgium. The goal of this discussion is to explore the
Not Dead Yet: Estrogen Deserves Another Chance
that leuprolide increased longevity in rats by 18 percent with 6 months of treatment. Mark Smith Craig,
The Pathogen Hypothesis—Challenging the Primacy of Genetics in Late-Onset Alzheimer Disease.
We invite you to participate in this "offline" Forum Discussion led by Brian Balin (Philadelphia College of Osteopathic Medicine). This discussion will extend our previously held live discussion on the Pathogen Hypothesis of Alzheimer Disease. T
Are Glia Active Participants in Neurodegenerative Disease?
Ben Barres led the first in a series of Alzforum discussions about the role of glial cells in Alzheimer’s and other neurodegenerative diseases. It has become trendy to say that glia are more than just glue, but in reality, science has not yet advanced sig
Differential Neuronal Vulnerability
dopamine neurons: implications for selective vulnerability in parkinsonism. Neurobiol Dis, 18, 19-31.
Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.
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