Protofibrillar Aβ in Alzheimer's Disease
Dean? William Klein: Let me start by saying how much I enjoyed the paper by Dean et al. A good read.
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Dean? William Klein: Let me start by saying how much I enjoyed the paper by Dean et al. A good read.
flow. (Editor’s note: see recent paper about connectivity data in young ApoE4 data: Filippini et al.,
inactivation. Vikram Khurana Dohoon, Li-Huei, congratulations on the great paper! Cdk5 seems to be a central
This live discussion was held on the paper by O'Doherty et al., in Science, on "An
2011). The papers followed a previous release of draft guidelines at the July 2010 ICAD conference in
molecule as a recent (Apr 18) Science paper mentions and the article by Charlie Glabe of 1994
Orlando, I suppose... I have read your recent paper, Chris, and am excited to have you join us. Chris Link
signalling. I thought that we might open the forum with a brief discussion about why papers have demonstrated
described in the Farrer paper (Farrer et al., 2001) is a partially active mutant as described in our Nature
your main concepts in the JAD papers. Suzanne de la Monte Our main concepts are that Alzheimer disease
Perry: Is that what you said in your earlier papers? Carl Cotman: Yes Carl Cotman: Cummings- get back to
it is a possibility (see related ARF news story). Changiz Geula Gunnar, in your paper you mention
thank Science Translational Medicine for giving Alzforum readers temporary free access to this paper
papers details potential benefits of the cholinesterase inhibitors, with the data for each being
some role in the nucleus? Tom Fagan All, there's a paper coming out in this week's Cell from
Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.