An Idiosyncratic View of AD Biomarkers
Dr. Coleman led this live discussion on how biomarker information and basic science of AD can
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Dr. Coleman led this live discussion on how biomarker information and basic science of AD can
Foundation), June Kinoshita (Alzheimer Research Forum). Note: Transcript has been edited for clarity and
Davies of Albert Einstein College of Medicine in the Bronx, New York, and Bart De Strooper at K.U.
from the Times. It was a lay piece and not broken down by compound. Craig Atwood My point being that
environmental influences of the microbial world in chronic diseases of aging cannot be dismissed. Time and
expressing familial Alzheimer disease (FAD)-linked amyloid-β precursor protein (APP) and presenilin (PS) that
Alzheimer’s and other neurodegenerative diseases. It has become trendy to say that glia are more than just
Casadesus et al. (.pdf) in the inaugural issue of Alzheimer's & Dementia. (Article and commentaries
development effort for SMA and the tech transfer process is consuming an inordinate amount of time. Jens
time points and we can get back to you within days with details. Rachael Neve But did you do any of the
would be clinically relevant? We could look with different severities and time durations as well as
Questions and Answers About Alzheimer's Disease? Thomas Wisniewski Tom, we wrote the book because of
capture and real-time polymerase chain reaction (PCR). Tom Fagan Ole, Vik, Chee Yeun, are these innate
Alzheimer's disease (Aβ), Parkinson's, Huntington's, Prion diseases and Type II diabetes can all
biomarker modalities in middle-aged folks and then following them over time. I'm working with Dr.
Have a topic idea for a webinar? We would love to hear it. Send an email to webinars [at] alzforum [dot] org.