Fishel MA, Watson GS, Montine TJ, Wang Q, Green PS, Kulstad JJ, Cook DG, Peskind ER, Baker LD, Goldgaber D, Nie W, Asthana S, Plymate SR, Schwartz MW, Craft S. Hyperinsulinemia Provokes Synchronous Increases in Central Inflammation and {beta}-Amyloid in Normal Adults. Arch Neurol. 2005 Aug 8; PubMed.

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  1. This paper raises several provocative issues. It also lends further support to those who believe that the secondary function of telomeres is to serve as a checkpoint for uncontrolled cellular proliferation. This makes sense in tissues that continue to undergo mitosis. But it raises the question, "What is the function of telomeres in non-replicative cells?" They must have some function; what is their function and why are they there? I have an idea but would like to hear from others first.

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  1. The Long and Short of It: Insulin Trumps Telomeres in Organismal Aging