Keith A. Crutcher Interviews John Trojanowski
Keith A. Crutcher Interviews John Trojanowski
ALL NEWS
6546 RESULTS
Sort By:
- Most Recent
- Most Commented
American Society for Cell Biology: 38th Annual Meeting
American Society for Cell Biology 38th Annual Meeting: Tau Protein in Neurodegenerative Diseases American Society for Cell Biology 38th Annual Meeting: More About Tau American Society for Cell Biology 38th Annual Meeting: Novel Protein Binds to APP and Mi
American Society for Cell Biology 38th Annual Meeting: More About Tau
Maas et al. (Abstract 2285) studied how phosphorylation of two different regions in tau proteins affects binding of tau protein to the plasma-membrane (PM). The two major phosphorylation sites studied...
American Society for Cell Biology 38th Annual Meeting, San Francisco, 12-16 December, 1998
A special interest subgroup meeting on 'Tau protein in Neurodegenerative Diseases' was organized by Gloria Lee, University of Iowa. This was a timely and stimulating meeting...
American Society for Cell Biology 38th Annual Meeting: Tau Protein in Neurodegenerative Diseases
A special interest subgroup meeting on "Tau protein in Neurodegenerative Diseases" was organized by Gloria Lee, University of Iowa. This was a timely and stimulating meeting that was energized in large part by the recent discovery...
American Society for Cell Biology 38th Annual Meeting: Novel Protein Binds to APP and Microtubules
Zheng et al. (Abstract 516) described the characterization of PAT1, a novel protein that binds to an 11-residue segment of the amyloid precursor protein (APP) in yeast 2-hybrid assays...
American Society for Cell Biology 38th Ann Mtg: Chaperone Clings to Aβ
Holtzman et al. (Abstract 612) reported that 95% of immunoreactive β-amyloid in human cerebral spinal fluid is bound to the ER chaperone Erp57...
American Society for Cell Biology 38th Annual Meeting: A Lethal Double Knockout
Harada et al. (Abstract 902) have now disrupted the MAP2 gene in mouse. Like tau and MAP1B knockout mice, MAP-2 knockout mice are fertile and have apparently normal brain cytoarchitecture...
American Society for Cell Biology 38th Annual Meeting: Glutamate Receptor Breakdown
Chan et al. (Abstract 1421) used immunoblot analysis to quantitatively demonstrate that the polypeptides corresponding to AMPA (GluR2 and 3) and NMDA subfamilies of the glutamate receptors are decreased in AD brain lysates...
American Society for Cell Biology 38th Annual Meeting: Presenilin-binding Proteins Abound
Mervyn Monteiro (the author of this report) and colleagues described the identification and characterization of two different proteins that interact with presenilins in the yeast-2 hybrid interaction trap (Y2H)...
American Society for Cell Biology 38th Annual Meeting: Presenilins Arrest Cell Cycle
Monteiro and Janicki et al. (Abstract 1057) showed by BrdU labeling of HeLa cells that overexpression of both PS1 and PS2 arrest cells in the G1 phase of the cell cycle...
June Kinoshita Interviews Bruce Yanker
June Kinoshita Interviews Bruce Yanker
An APP-Presenilin Connection
Mutations in the β-amyloid precursor protein (APP), presenilin-1 and presenilin-2 cause inherited forms of Alzheimer's disease, but how do these protein interact, normally and in the disease process?...
What FTDP-17 Mutations Do
A new study elucidates the molecular mechanisms that may explain how mutations of the tau gene give rise to frontotemporal dementia and parkinsonism (FTDP-17), an inherited disorder that is similar to Alzheimer's...
Hungarian Conference on Alzheimer's Disease and Related Disorders 1998
News Coverage of the 4th Hungarian Conference on AD: Hungarian Conference on Alzheimer's Disease and Related Disorders 1998
Current Filters
- Date Range : All x