PAPER Fei F, Jie B, Zhang D
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329967 RESULTS
Lotta Granholm on Domains of genome-wide gene expression dysregulation in Down's syndrome.
COMMENT The study suggested that nuclear compartments of trisomic cells undergo modifications of the chromatin environment influencing the overall transcriptome, and that gene expression dysregulation domains (GEDDs) may therefore contribute to phenotypic changes
PAPER Hu F, Liu F
Targeting tissue-specific metabolic signaling pathways in aging: the promise and limitations.
Protein Cell. 2014 Jan;5(1):21-35. Epub 2014 Jan 29 PubMed: 24474199Maria Hanford
Non-profit organization Forest Hills, United States
PAPER Diez-Fraile A, Lammens T, Tilleman K, Witkowski W, Verhasselt B, De Sutter P, Benoit Y, Espeel M, D'Herde K
Age-associated differential microRNA levels in human follicular fluid reveal pathways potentially determining fertility and success of in vitro fertilization.
Hum Fertil (Camb). 2014 Jun;17(2):90-8. Epub 2014 Mar 31 PubMed: 24684237PAPER Buga AM, Margaritescu C, Scholz CJ, Radu E, Zelenak C, Popa-Wagner A
Transcriptomics of post-stroke angiogenesis in the aged brain.
Front Aging Neurosci. 2014;6:44. Epub 2014 Mar 18 PubMed: 24672479CNS Partnering and Deal-Making Conference, 2014
CONFERENCE 2014-09-18 00:00:00-2014-09-19 00:00:00 San Francisco, California 18 September 2014 to 19 September 2014 View Conference Website 1
Jenifer Lee
University of California, IrvineIrvine, United States
Keystone Symposium: Alzheimer’s Disease Beyond Aβ
CONFERENCE 2010-01-10 00:00:00-2010-01-15 00:00:00 Copper Mountain, Colorado 10 January 2010 to 15 January 2010 0
Louise Walker
Alzheimer's Research UKCambridge, United Kingdom
Emma O'Brien
United Kingdom
Jessica Martinez
N/A
Lique Du
Tg4-42
RESEARCH MODELS Summary The Tg4-42 model was engineered to produce the peptide Aβ 4-42, one of a variety of N-terminally truncated Aβ species identified in the AD brain. The Aβ 4-42 peptide, which begins with a phenylalanine at position 4, may be a particularly abundant
TBA42
RESEARCH MODELS Summary The TBA42 model was designed to produce and secrete an N-terminally truncated Aβ peptide. TBA42, which stands for truncated beta-amyloid 42, specifically encodes an Aβ 3–42 peptide with a glutamine substituted for the naturally occurring glutamate
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