. Hyperinsulinemia Provokes Synchronous Increases in Central Inflammation and {beta}-Amyloid in Normal Adults. Arch Neurol. 2005 Aug 8; PubMed.

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  1. This paper raises several provocative issues. It also lends further support to those who believe that the secondary function of telomeres is to serve as a checkpoint for uncontrolled cellular proliferation. This makes sense in tissues that continue to undergo mitosis. But it raises the question, "What is the function of telomeres in non-replicative cells?" They must have some function; what is their function and why are they there? I have an idea but would like to hear from others first.

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