All Comments by
- Bradley Hyman on SNPs associated with cerebrospinal fluid phospho-tau levels influence rate of decline in Alzheimer's disease.
- Jack T. Rogers on Iron-export ferroxidase activity of β-amyloid precursor protein is inhibited by zinc in Alzheimer's disease.
- Virgil Muresan on Soluble amyloid precursor protein (APP) regulates transthyretin and Klotho gene expression without rescuing the essential function of APP.
- Melinda R. Severance-Fengel on Where in the World Are the iPS Cells?
- Tohru Hasegawa on Brain Aβ Patterns Linked to Brain Energy Metabolism
- Makoto Kuro-o on Soluble amyloid precursor protein (APP) regulates transthyretin and Klotho gene expression without rescuing the essential function of APP.
- Eric M. Reiman on Brain Aβ Patterns Linked to Brain Energy Metabolism
- William Klunk on Brain Aβ Patterns Linked to Brain Energy Metabolism
- Margaret Fahnestock on Oligomeric amyloid-{beta} inhibits the proteolytic conversion of brain-derived neurotrophic factor (BDNF), AMPA receptor trafficking, and classical conditioning.
- David Holtzman on Diagnosis-independent Alzheimer disease biomarker signature in cognitively normal elderly people.
- Virgil Muresan on Lilly Halts IDENTITY Trials as Patients Worsen on Secretase Inhibitor
- Jürgen Götz on Homocysteine-lowering by B vitamins slows the rate of accelerated brain atrophy in mild cognitive impairment: a randomized controlled trial.
- Tony Wyss-Coray on A serum protein-based algorithm for the detection of Alzheimer disease.
- Akihiko Nunomura on Iron Export? New Role Links APP, Metals, to Oxidative Stress
- Jürgen Götz on Neurotoxicity of Alzheimer's disease Aβ peptides is induced by small changes in the Aβ42 to Aβ40 ratio.