All Comments by Takaomi Saido

  1. The Arctic AβPP mutation leads to Alzheimer's disease pathology with highly variable topographic deposition of differentially truncated Aβ.
  2. The Off-Rate of Monomers Dissociating from Amyloid-β Protofibrils.
  3. Brain amyloid-β oligomers in ageing and Alzheimer's disease.
  4. Fluorine-18 radiolabeled heterocycles as PET tracers for imaging β-amyloid plaques in Alzheimer's disease.
  5. A transgenic Alzheimer rat with plaques, tau pathology, behavioral impairment, oligomeric aβ, and frank neuronal loss.
  6. Comparing PET imaging and CSF measurements of Aß
  7. Alzheimer's disease neurodegenerative biomarkers are associated with decreased cognitive function but not β-amyloid in cognitively normal older individuals.
  8. Physiologic brain activity causes DNA double-strand breaks in neurons, with exacerbation by amyloid-β.
  9. Oligomers, fact or artefact? SDS-PAGE induces dimerization of β-amyloid in human brain samples.
  10. PET Scanning of Brain Tau in Retired National Football League Players: Preliminary Findings.
  11. Membrane fusion and vesicular transformation induced by Alzheimer's amyloid beta.
  12. Diverging patterns of amyloid deposition and hypometabolism in clinical variants of probable Alzheimer's disease.
  13. Isomerase Pin1 stimulates dephosphorylation of tau protein at cyclin-dependent kinase (Cdk5)-dependent Alzheimer phosphorylation sites.
  14. α-Synuclein membrane association is regulated by the Rab3a recycling machinery and presynaptic activity.
  15. Hypoxia Increases Aβ-Induced Tau Phosphorylation by Calpain and Promotes Behavioral Consequences in AD Transgenic Mice.