All Comments by John Trojanowski

  1. A century of Alzheimer's disease.
  2. Chronic lithium administration to FTDP-17 tau and GSK-3beta overexpressing mice prevents tau hyperphosphorylation and neurofibrillary tangle formation, but pre-formed neurofibrillary tangles do not revert.
  3. A peptide zipcode sufficient for anterograde transport within amyloid precursor protein.
  4. Reduction of soluble Abeta and tau, but not soluble Abeta alone, ameliorates cognitive decline in transgenic mice with plaques and tangles.
  5. Double Paper Alert—A Function for BACE, a Basis for Amyloid
  6. HDDD2 is a familial frontotemporal lobar degeneration with ubiquitin-positive, tau-negative inclusions caused by a missense mutation in the signal peptide of progranulin.
  7. Amyloid-beta peptide remnants in AN-1792-immunized Alzheimer's disease patients: a biochemical analysis.
  8. Dangerous Liaisons—Tau and Aβ, Together at Last?
  9. Is It Good for You? Amyloid Shows New Side in Mammalian Cells
  10. Mouse of a Different Color: Presenilin Mutations Affect Melanin
  11. Clinical and pathological characterization of progressive aphasia.
  12. Formation of tau inclusions in knock-in mice with familial Alzheimer disease (FAD) mutation of presenilin 1 (PS1).
  13. Longitudinal CSF and MRI biomarkers improve the diagnosis of mild cognitive impairment.
  14. Anchorless prion protein results in infectious amyloid disease without clinical scrapie.
  15. The cell cycle in Alzheimer disease: a unique target for neuropharmacology.