All Comments by M. Flint Beal

  1. Could Disposing of Damaged Mitochondria Treat Alzheimer’s Disease?
  2. It’s MAGIC: Yeast Mitochondria Make Cytosolic Protein Aggregates Disappear
  3. Preventive methylene blue treatment preserves cognition in mice expressing full-length pro-aggregant human Tau.
  4. Amyloid-β peptide induces mitochondrial dysfunction by inhibition of preprotein maturation.
  5. Protein Destroying Muscle, Bone, Nerves Parks on Mitochondria
  6. Kynurenine 3-monooxygenase inhibition in blood ameliorates neurodegeneration.
  7. Impaired mitochondrial dynamics and abnormal interaction of amyloid beta with mitochondrial protein Drp1 in neurons from patients with Alzheimer's disease: implications for neuronal damage.
  8. Hearing Loss Linked to Incident Dementia
  9. Inhibition of amyloid-beta (Abeta) peptide-binding alcohol dehydrogenase-Abeta interaction reduces Abeta accumulation and improves mitochondrial function in a mouse model of Alzheimer's disease.
  10. Mitochondrial bioenergetic deficit precedes Alzheimer's pathology in female mouse model of Alzheimer's disease.
  11. Mitochondrial Gridlock—Syntaphilin Puts Brakes on Axonal Transport
  12. Progressive parkinsonism in mice with respiratory-chain-deficient dopamine neurons.
  13. DNA Deletions Sap Mitochondria in Parkinson Neurons
  14. A cluster of metabolic defects caused by mutation in a mitochondrial tRNA.
  15. Bile Acid Proves Neuroprotective in Huntington's Model