All Comments by Rudolf Bloechl
- Research Brief: Blocking Apoptosis Delays ALS in Mice
- Honolulu: The Missing Link? Tau Mediates Aβ Toxicity at Synapse
- Deleterious effects of amyloid beta oligomers acting as an extracellular scaffold for mGluR5.
- Caspase-3 activation via mitochondria is required for long-term depression and AMPA receptor internalization.
- Cell-produced alpha-synuclein is secreted in a calcium-dependent manner by exosomes and impacts neuronal survival.
- Molecular cross talk between misfolded proteins in animal models of Alzheimer's and prion diseases.
- The p75 neurotrophin receptor promotes amyloid-beta(1-42)-induced neuritic dystrophy in vitro and in vivo.
- p75 reduces beta-amyloid-induced sympathetic innervation deficits in an Alzheimer's disease mouse model.
- Peripheral p75 Protects Against Aβ Toxicity
- Selective vulnerability in Alzheimer's disease: amyloid precursor protein and p75(NTR) interaction.
- Keystone: Partners in Crime—Do Aβ and Prion Protein Pummel Plasticity?