All Comments by Alexei Koudinov
- What evidence would prove the amyloid hypothesis? Towards rational drug treatments for Alzheimer's disease.
- Beta-secretase protein and activity are increased in the neocortex in Alzheimer disease.
- Explaining the cause of the amyloid burden in Alzheimer disease.
- Characterization of neuronal dystrophy induced by fibrillar amyloid beta: implications for Alzheimer's disease.
- Continuing strategies for inhibiting Alzheimer's gamma-secretase.
- Human antibodies against amyloid beta peptide: a potential treatment for Alzheimer's disease.
- Paradigm shifts in Alzheimer's disease and other neurodegenerative disorders: the emerging role of oligomeric assemblies.
- Neuroscience. Long-awaited technique spots Alzheimer's toxin.
- The amyloid hypothesis of Alzheimer's disease: progress and problems on the road to therapeutics.
- Alzheimer beta-amyloid peptides: structures of amyloid fibrils and alternate aggregation products.
- Alzheimer's Disease: Genes, Pathogenesis and Risk Prediction.
- Peptide aggregation in neurodegenerative disease.
- Abeta42 plasma in the diagnosis of Alzheimer's disease.
- Tau, phospho-tau and Ab42 in cerebrospinal fluid in Alzheimer's disease.
- Cerebrospinal fluid 24S-hydroxycholesterol is increased in Alzheimer's disease compared to healthy controls.