All Comments by Inez Vincent
- Apolipoprotein E and Reelin ligands modulate tau phosphorylation through an apolipoprotein E receptor/disabled-1/glycogen synthase kinase-3beta cascade.
- Ectopic expression of cell cycle markers in models of induced programmed cell death in dopamine neurons of the rat substantia nigra pars compacta.
- Oxidative Stress Triggers Neuronal Cell-Cycle Reentry
- p53 as Mediator of Oxidative Stress and Inflammation?
- Cyclin D1 and cyclin E are co-localized with cyclo-oxygenase 2 (COX-2) in pyramidal neurons in Alzheimer disease temporal cortex.
- Nuclear localization of N-terminal mutant huntingtin is cell cycle dependent.
- p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.
- p35/Cdk5 pathway mediates soluble amyloid-beta peptide-induced tau phosphorylation in vitro.
- Prominent neurodegeneration and increased plaque formation in complement-inhibited Alzheimer's mice.
- Cyclin-dependent kinases and P53 pathways are activated independently and mediate Bax activation in neurons after DNA damage.
- Reduced protein phosphatase 2A activity induces hyperphosphorylation and altered compartmentalization of tau in transgenic mice.
- Where do Alzheimer's plaques and tangles come from? Aging-induced protein degradation inefficiency.
- Loss and atrophy of layer II entorhinal cortex neurons in elderly people with mild cognitive impairment.
- Metabolic, metallic, and mitotic sources of oxidative stress in Alzheimer disease.
- Accumulation of C-terminally truncated tau protein associated with vulnerability of the perforant pathway in early stages of neurofibrillary pathology in Alzheimer's disease.