All Comments by Ming Chen
- Presenilins form ER Ca2+ leak channels, a function disrupted by familial Alzheimer's disease-linked mutations.
- Presenilin Loss of Function—Plan B for AD?
- After 40, DNA Damage Accrues in Genes, Hampering Expression
- Gonadal Hormone Withdrawal, Apoptosis, and Generation of Beta Amyloid Peptide: A Vicious Cycle of Amyloidogenesis and Neurotoxicity in Alzheimer's Disease
- Gonadal Hormone Withdrawal, Apoptosis, and Generation of Beta Amyloid Peptide: A Vicious Cycle of Amyloidogenesis and Neurotoxicity in Alzheimer's Disease
- Calcium Signaling Deficit in the Origin of Alzheimer's Disease
- Nomenclature Discussion
- Calcium Signaling Deficit in the Origin of Alzheimer's Disease
- Calcium Signaling Deficit in the Origin of Alzheimer's Disease
- Calcium Signaling Deficit in the Origin of Alzheimer's Disease
- Nomenclature Discussion